Tuesday , August 16 2022

New pneumonia has been discovered



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CODD and colitis, such as blood circulation, are a major threat to public health.

Such conditions are the result of excessive immune system.

Therefore, the discovery of molecules is awakening 5487 Strike. Molecules have the potential to forbid a protein that plays an important role in molecular diseases.

Molecular blocking pneumonia in mice

Researchers found the researchers at Stockholm, Stockholm University, Texas University, NNTU and Zinteph in Trondheim.

The start button for an overreaction

Response from your immune system is a bloat aimed at target or injury. But some are swollen:

Many people with acute myoclonic signals provide a lot of signal roses to the body. This is a short name for "Active Oxygen Species." The legacy of the cell, the inflammation of the DNA begins and destroys.

And the OGG1 comes on the pants. Enzyme is an encouraging body that promotes body reaction.

DNA repairing damages in our cells have OGG1. The immune system is a beginning.

This is the Start button that enhances the immune system for people with automatic illness. The immune system is a disease that starts to attack healthy cells.

Repairs and enzymes are stronger.

Earlier studies have shown that mice without OGG1 may not be able to perform a strong immune system, so the normal mice get much faster than normal mice.

Five years ago, Zinteph's researcher Tornwood was interested in this enzyme when he worked at the Carolinskaya Institute (KI).

He and his colleagues will develop a rich, preventing chemicals from repairs.

It is not clear that the damage to DNA, which is going to repair the enzyme,

– We thought Ross was destroyed accidentally in the past. But now these changes do not seem to be bad for everyone. However, Vinci describes part of a normal immune response. But this needs more research.

This is how molecules work. If the DNA is found, the repair enzyme tricks to believe it. It does not make it so much swollen. (Illustration: Zintaf)
This is how molecules work. If the DNA is found, the repair enzyme tricks to believe it. It does not make it so much swollen. (Illustration: Zintaf)
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This is how molecules work. If the DNA is found, the repair enzyme tricks to believe it. It does not make it so much swollen. (Illustration: Zintaf)

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Enzyme is amazing

In 2013, the research team began hunting for the opportunity to activate OGG1.

"The idea was to investigate a chemical product that was supposed to keep the protein protein to" disarm "the use of this drug: it has already been wasted by the protein that destroys DNA. Hey.

The researcher and biochemist Torkild Wins in the lab in Sintef. (Photo: Thor Nielsen)
The researcher and biochemist Torkild Wins in the lab in Sintef. (Photo: Thor Nielsen)
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The researcher and biochemist Torkild Wins in the lab in Sintef. (Photo: Thor Nielsen)

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"I've done a precaution to measure the activity in this kind of enzyme, but with many occupational methods," says Vince.

– Through fluorosis, Enzyme has developed a method to read functionality – ie DNA-Luminees – we can read thousands of samples in a few hours.

OGG1 disabled drugs have been abandoned after inspecting 18,000 items for repairs. It took a year.

Development of molecular labs

A hard process started to improve that structure, and thus all the necessary materials to function in a living cell.

For two years the researchers made a thousand differences in objects. In the end, they told the youngsters, "T5486", because the drug was given by the name TH5487.

This medication has proven three important properties to investigate researchers:

1. Infection The protein was in OGG1, so it could not be connected to digestive and DNA.

2. OGG1 can also be deactivated in living cells

3. In animals animals were very stable.

New rat in Texas

The researchers at the University of Texas examined the mice of the lungs and researched.

TH5487 can help prevent maintenance enzyme from their job quickly and efficiently. Hence, the inflammation began to cure the immune system and the lungs.

So pneumonia is eager. Hence the research team believes they have experienced a new incidence of swelling and can be used or replaced by existing therapies.

"The aim is to make a drug running in human beings, it's too long, the laws are complex and the costs are great and believe it's important to take care of yourself from the immune system and blood sugar."

Reference:

Winsnes, TEMA: The OGG1's small malfunction of inhaleric prophylaxional suppresses gene expression and inflammation. Science. (2018) (summary) DOI: 10.1126 / science.aar8048

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